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Subclinical EEG abnormalities, including epileptiform or non-specific discharges, are frequently reported in ASD and are considered markers of altered cortical excitability or neurobiological vulnerability rather than epilepsy per se

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Upon neuronal depolarization, methylnorepinephrine is released into the synaptic cleft, where it preferentially activates presynaptic 2adrenergic receptors rather than postsynaptic 1receptors ( panel 4 )

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